Amyloid Beta
Amyloid beta (Aβ) is a peptide implicated in Alzheimer's disease and other forms of dementia. It forms when amyloid precursor protein (APP) is improperly cleaved by enzymes. Accumulating Aβ peptides aggregate into plaques, a hallmark of Alzheimer's pathology. These plaques disrupt cell function, trigger inflammation, and promote neuronal death, contributing to cognitive decline and memory loss. Research aims to understand the mechanisms of Aβ formation and aggregation, seeking therapeutic strategies to prevent or reduce plaque buildup. Despite its central role in Alzheimer’s, the exact relationship between amyloid beta and disease progression remains complex, involving genetic, environmental, and biochemical factors.
Committee Members
Jun Hua
Johns Hopkins University , United States
Nazneen N Dewji
Cenna Biosciences Inc, United States
Jacqueline London
Paris Diderot University, FranceSubmit your abstract Today
Title : Neuroimaging-based evaluation of scalp acupuncture for neural repair and reorganization in children with cerebral palsy
Zhenhuan Liu, Guangzhou University of Chinese Medicine, China
Title : Neuromodulation of scalp electroacupuncture in the treatment of autism spectrum disorder
Zhenhuan Liu, Guangzhou University of Chinese Medicine, China
Title : Make a difference
Jacqueline Tuppen, Cogs Club, United Kingdom
Title : Association between depressive symptoms and cognitive impairment severity in a real-world memory clinic cohort
Yun Ju Hsieh, Taipei City Hospital, Taiwan
Title : Health care professionals in Somaliland had low levels of knowledge and attitudes towards dementia care: A pilot survey
Mohamed Abdilahi Duale, Somali Red Crescent Society, Somalia
Title : Imaging brain clearance in Alzheimer’s disease
Jun Hua, Johns Hopkins University , United States