Dementia, including Alzheimer's Disease, arises from complex pathogenic processes in the brain. In Alzheimer's Disease, abnormal protein deposits, such as beta-amyloid plaques and tau tangles, disrupt neuronal communication and lead to cell death. These changes primarily affect regions involved in memory and cognition, gradually impairing cognitive functions like reasoning, memory recall, and behavior control. Additionally, inflammation, oxidative stress, and genetic factors contribute to the disease's progression. Early symptoms often include forgetfulness and difficulty with familiar tasks, progressing to profound memory loss and personality changes. Understanding these mechanisms is crucial for developing therapies aimed at slowing or halting the progression of dementia, addressing both the cognitive and emotional challenges faced by individuals and their caregivers.
Jun Hua
Johns Hopkins University , United States
Nazneen N Dewji
Cenna Biosciences Inc, United States
Jacqueline London
Paris Diderot University, FranceSubmit your abstract Today
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